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This Levaquin crap ruined Karen's future!

Levofloxacin (Levaquin), Ciprofloxacin (Cipro), Moxifloxacin (Avelox), Norfloxacin (Noroxin), Ofloxacin (Floxin), Gemifloxacin (Factive) and Finafloxacin (Xtoro) should be taken off the market, and the FDA knows it! Levaquin made it so she could not walk from the tendinitis that it caused.

It is my conjecture that this class of antibiotics are damaging the spinal Dura, what contains the spinal fluid, so that the treatments for her CSF Leaks did not work. There is ZERO study on this FQ drug/Dura connection.

I will be at the first ever Intracranial Hypotension Symposium this fall to discuss this with the worlds leading CSF Leak doctors.

Based on my conjecture and input Lisa Bloomquist Palmer of Floxie Hope did her own homework and wrote: Do Fluoroquinolones Cause Cerebrospinal Fluid Leaks?

Karen's medical records from 2003 when she was given Levaquin. I'm posting them in the hopes they will help someone.

Karen spent a year crawling around the house on this construction, skate board like thing so her ankles would heal:

All of this crap is the result of these #)$*#)$*# drugs!:

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The Karen Paddock Story at Pain Nation with Ken McKim

The condensed version of Karen's life of pain. Antibiotic issue is covered in the last ten minutes.

http://www.newsnet5.com/news/local-news/investigations/levaquin-fda-fails-to-disclose-additional-serious-side-effects-of-antibiotic-linked-to-deaths is my Cleveland/Akron TV interview. 3,000 dead. 200,000 injured by these drugs per the FDA. That is estimated to be only one-percent of the real numbers!

The News Net5 FDA Report.

For Immediate Release July 26, 2016

Release: The U.S. Food and Drug Administration today approved safety labeling changes for a class of antibiotics, called fluoroquinolones, to enhance warnings about their association with disabling and potentially permanent side effects and to limit their use in patients with less serious bacterial infections.

“Fluoroquinolones have risks and benefits that should be considered very carefully,” said Edward Cox, M.D., director of the Office of Antimicrobial Products in the FDA’s Center for Drug Evaluation and Research. “It’s important that both health care providers and patients are aware of both the risks and benefits of fluoroquinolones and make an informed decision about their use.”

Fluoroquinolones are antibiotics that kill or stop the growth of bacteria. While these drugs are effective in treating serious bacterial infections, an FDA safety review found that both oral and injectable fluroquinolones are associated with disabling side effects involving tendons, muscles, joints, nerves and the central nervous system. These side effects can occur hours to weeks after exposure to fluoroquinolones and may potentially be permanent.

Because the risk of these serious side effects generally outweighs the benefits for patients with acute bacterial sinusitis, acute exacerbation of chronic bronchitis and uncomplicated urinary tract infections, the FDA has determined that fluoroquinolones should be reserved for use in patients with these conditions who have no alternative treatment options. For some serious bacterial infections, including anthrax, plague and bacterial pneumonia among others, the benefits of fluoroquinolones outweigh the risks and it is appropriate for them to remain available as a therapeutic option.

FDA-approved fluoroquinolones include levofloxacin (Levaquin), ciprofloxacin (Cipro), ciprofloxacin extended-release tablets, moxifloxacin (Avelox), ofloxacin and gemifloxacin (Factive). The labeling changes include an updated Boxed Warning and revisions to the Warnings and Precautions section of the label about the risk of disabling and potentially irreversible adverse reactions that can occur together. The label also contains new limitation-of-use statements to reserve fluoroquinolones for patients who do not have other available treatment options for acute bacterial sinusitis, acute bacterial exacerbation of chronic bronchitis and uncomplicated urinary tract infections. The patient Medication Guide that is required to be given to the patient with each fluoroquinolone prescription describes the safety issues associated with these medicines.

The FDA first added a Boxed Warning to fluoroquinolones in July 2008 for the increased risk of tendinitis and tendon rupture. In February 2011, the risk of worsening symptoms for those with myasthenia gravis was added to the Boxed Warning. In August 2013, the agency required updates to the labels to describe the potential for irreversible peripheral neuropathy (serious nerve damage).

In November 2015, an FDA Advisory Committee discussed the risks and benefits of fluoroquinolones for the treatment of acute bacterial sinusitis, acute bacterial exacerbation of chronic bronchitis and uncomplicated urinary tract infections based on new safety information. The new information focused on two or more side effects occurring at the same time and causing the potential for irreversible impairment. The advisory committee concluded that the serious risks associated with the use of fluoroquinolones for these types of uncomplicated infections generally outweighed the benefits for patients with other treatment options.

Today’s action also follows a May 12, 2016, drug safety communication advising that fluoroquinolones should be reserved for these conditions only when there are no other options available due to potentially permanent, disabling side effects occurring together. The drug safety communication also announced the required labeling updates to reflect this new safety information.

The FDA, an agency within the U.S. Department of Health and Human Services, protects the public health by assuring the safety, effectiveness, security of human and veterinary drugs, vaccines and other biological products for human use, and medical devices. The agency is also responsible for the safety and security of our nation’s food supply, cosmetics, dietary supplements, products that give off electronic radiation, and for regulating tobacco products.

### – http://www.fda.gov/NewsEvents/Newsroom/PressAnnouncements/ucm513183.htm

Please see: Our Win at the FDA hearing on Fluoroquinolone Antibiotics for details and videos of the hearing at the FDA on November 5th 2015.

FDA Hearing meeting brief and the 617 page PDF report where the the FDA identified a syndrome associated with fluoroquinolone toxicity—one that “floxies” have been pushing for recognition of for years. It is called Fluoroquinolone Associated Disability (FQAD).

EMedExpert gives us this list of Fluoroquinolone Antiboitics:

Generic	Brand Name
First Generation
Flumequine	Flubactin
Nalidixic acid	NegGam, Wintomylon
Oxolinic acid	Uroxin
Piromidic acid	Panacid
Pipemidic acid	Dolcol
Rosoxacin	Eradacil
Second Generation
Ciprofloxacin	Cipro, Cipro XR, Ciprobay, Ciproxin
Enoxacin	Enroxil, Penetrex
Lomefloxacin	Maxaquin
Nadifloxacin	Acuatim, Nadoxin, Nadixa
Norfloxacin	Lexinor, Noroxin, Quinabic, Janacin
Ofloxacin	Floxin, Oxaldin, Tarivid
Pefloxacin	Peflacine
Rufloxacin	Uroflox
Third Generation
Balofloxacin	Baloxin
Gatifloxacin	Tequin, Zymar
Grepafloxacin	Raxar
Levofloxacin	Cravit, Levaquin
Moxifloxacin	Avelox, Vigamox
Pazufloxacin	Pasil, Pazucross
Sparfloxacin	Zagam
Temafloxacin	Omniflox
Tosufloxacin	Ozex, Tosacin
Fourth Generation
Besifloxacin	Besivance
Clinafloxacin
Gemifloxacin	Factive
Sitafloxacin	Gracevit
Trovafloxacin	Trovan
Prulifloxacin	Quisnon

Sadly new ones are being added all of the time. Five of the older generations were already pulled from the market due to the dangers they presented.

How many doctors have actually read the TEN PAGES of warnings in this 67 page document about Levaquin?

http://www.levaquin.com/sites/default/files/pdf/levaquin.pdf

There is a new drug, Otezla (Apremilast), to treat psoriatic arthritis out. There have been reports that it works the same way as the above drugs, and has been attacking tendons. Not something I'd personally be taking after seeing what the drugs did to Karen.

FDA Drug Safety Communication: FDA requires label changes to warn of risk for possibly permanent nerve damage from antibacterial fluoroquinolone drugs taken by mouth or by injection:

Safety Announcement

[8-15-2013] The U.S. Food and Drug Administration (FDA) has required the drug labels and Medication Guides for all fluoroquinolone antibacterial drugs be updated to better describe the serious side effect of peripheral neuropathy. This serious nerve damage potentially caused by fluoroquinolones (see Table for a list) may occur soon after these drugs are taken and may be permanent.

The risk of peripheral neuropathy occurs only with fluoroquinolones that are taken by mouth or by injection. Approved fluoroquinolone drugs include levofloxacin (Levaquin), ciprofloxacin (Cipro), moxifloxacin (Avelox), norfloxacin (Noroxin), ofloxacin (Floxin), and gemifloxacin (Factive). The topical formulations of fluoroquinolones, applied to the ears or eyes, are not known to be associated with this risk.

If a patient develops symptoms of peripheral neuropathy, the fluoroquinolone should be stopped, and the patient should be switched to another, non-fluoroquinolone antibacterial drug, unless the benefit of continued treatment with a fluoroquinolone outweighs the risk. Peripheral neuropathy is a nerve disorder occurring in the arms or legs. Symptoms include pain, burning, tingling, numbness, weakness, or a change in sensation to light touch, pain or temperature, or the sense of body position. It can occur at any time during treatment with fluoroquinolones and can last for months to years after the drug is stopped or be permanent. Patients using fluoroquinolones who develop any symptoms of peripheral neuropathy should tell their health care professionals right away.

FDA will continue to evaluate the safety of drugs in the fluoroquinolone class and will communicate with the public again if additional information becomes available.

Other fluoroquinolones related publications and sites:

https://onedrive.live.com/view.aspx?cid=B0735325185D9D26&resid=B0735325185D9D26%21149&app=Word

Dear Doctor, As you are probably aware, the fluoroquinolone class of antibiotics is useful for certain serious infections. Unfortunately, fluoroquinolones also have a long history of serious adverse drug reactions, many of them long term . (1) As a consequence of these reactions, several of these drugs have been removed from clinical practice or their use severely restricted. Besides the severe life threatening immediate reactions, those of a more chronic nature may occur.

The spectrum of these adverse reactions is extremely broad. Patients suffering from these reactions are often misdiagnosed, referred for a psychiatric consult or even unfairly labeled as “difficult patients.”

Many physicians have not been properly educated about the severe nature of these chronic adverse reactions, some of which result in life-long disabilities. Post-marketing studies of several flouroquinolones have shown an incidence of adverse reactions much higher than were originally reported in pre-clinical studies. (1,2,3)

You are probably aware that the fluoroquinolones are eukaryotic DNA gyrase and topoisomerase inhibitors very similar to many antineoplastic agents. Because of their similar mechanisms of action, it's no surprise that fluoroquinolones and many antineoplastic agents share similar toxicity profiles. Studies have even been conducted using fluoroquinolones to inhibit neoplastic chondrocyte growth in chondrosarcoma. (4)

There are many patients who have a syndrome of associated symptoms that include, but are not limited to: CNS agitation, depression, insomnia, new-onset anxiety and panic attacks, and even elevated intracranial pressure and visual abnormalities. They may also present with peripheral neuropathy usually of the small fiber type with temperature and pain sensory aberrations, but also often involving larger sensory and motor nerves. Spontaneous muscle activity with fasciculations, myokymia and myoclonic jerks may also occur. Many have musculoskeletal damage with degeneration of cartilage and tendons often leading to tendon rupture and severe ongoing musculoskeletal pain long after therapy has been discontinued. (1,2,3,4,5,6,7,8)

This complex symptomatology does not usually resolve after discontinuation of the inducing fluoroquinolone and may in fact worsen. Many patients go on to have disability that may persist for years. (1) Unfortunately, such patients are often seen by many physicians from multiple specialties who, given the complex symptomatology, fail to recognize a unifying diagnosis.

The mechanism of injury is not fully apparent, but several studies have been conducted and researchers have implicated the following possible mechanisms:

1. Inhibition or disruption of the CNS GABA receptor. (9)

2. Depletion of magnesium and disruption of cellular enzymatic function. (10)

3. Disruption of mitochondrial function and energy production. (11,12)

4. Oxidative injury and cellular death. (14)

This seems to be a functional disorder and structural abnormalities are not usually seen on radiological studies. (13) Patients may have abnormal EMG/NCV studies, abnormal skin punch neurologic density and morphology, abnormal vasomotor and sudomotor function on autonomic testing, and abnormal degeneration of tendons and cartilage on MRI. (13)

There may be a large number of these patients with coexisting endocrine abnormalities including: antithyroid antibodies and abnormal thyroid function, abnormal adrenal function with either hyper or hypocortisolism, hypogonadism, hypo or hyperglycemia and possibly impaired pituitary function. (13)

Most patients suffering from these side effects have a very clear onset of symptoms temporally related to a course of fluoroquinolone antibiotic. (13) They were often given the fluoroquinolone in conjunction with a corticosteroid or NSAID. Both of these classes of medications are associated with an increased incidence of adverse drug reaction from fluoroquinolones. (10,13)

As of yet no scientifically proven effective treatment is known, however patients will definitely benefit from your caring support and appropriate informed care. Of course, other diseases with similar symptoms need to be carefully ruled out.

There exists a large community of these patients who share information on the World Wide Web. Their numbers grow as the prescription of fluoroquinolones increases. Many of these patients are professionals like myself who have been affected by these drugs. Thank you for your time and consideration.

Todd R. Plumb MD

References:

1. Cohen JS; Peripheral Neuropathy Associated With Fluoroquinolones Annals of Pharmacotherapy. 2001;35(12):1540-1547

2. Francesca Lunzer Kritz; New Cipro, Same Side Effects, Washington Post, December 24, 2002.

3. Shepard CW et al; Antimicrobial Postexposure Prophylaxis for Anthrax: Adverse Events and Adherence Emerging Infectious Diseases ¡E Vol. 8, No. 10, October 2002

4. Fox EJ et al; The effects of ciprofloxacin and paclitaxel on metastatic and recurrent chondrosarcoma COMMUNITY ONCOLOGY ¡½ November/December 2005

5. Physisicans Desk Referfence 2006

6. de Bazignan DA etal; Psychiatric adverse effects of fluoroquinolone: review of cases from the French pharmacologic surveillance database[Article in French] Rev Med Interne. 2006 Jun;27(6):448-52. Epub 2006 Mar 9

7. FDA Medical Bulletin * October 1996 * Volume 26 Number 3 REPORTS OF ADVERSE EVENTS WITH FLUOROQUINOLONES

8. Saint F. etal; Tendinopathy associated with fluoroquinolones: individuals at risk, incriminated physiopathologic mechanisms, therapeutic management [Article in French] Prog Urol. 2001 Dec;11(6):1331-4.

9. De Sano A. etal; Adverse Reactions to Fluoroquinolones. An Overview on Mechanistic Aspects Current Medicinal Chemistry 2001, 8, 371-384 371

10. Stahlmann R. etal; Effects of magnesium deficiency on joint cartilage in immature Beagle dogsimmunohistochemistry, electron microscopy, and mineral concentrations, Archives of Toxicology. Jan. 2000 73(11,12)

11. Hayem G. Cytofluorometric analysis of chondrotoxicity of fluoroquinolone antimicrobial agents. Antimicrob Agents Chemother. 1994 Feb;38(2):243- 7.

12. Kozie[lstrok]; Ciprofloxacin reduces mitochondrial potential and inhibits calcium entry into Jurkat cells R European Journal of Biochemistry 2003; 1 Supplement 1 July: Abstract number: P4.8-33., Zab[lstrok]ocki K., Szczepanowska

13. http://health.groups.yahoo.com/group/quinolones/

14. Simonin MA etal. Pefloxacin-Induced Achilles Tendon Toxicity in Rodents: Biochemical Changes in Proteoglycan Synthesis and Oxidative Damage to CollagenAntimicrobial Agents and Chemotherapy, April 2000, p. 867-872, Vol. 44, No. 4

Note to readers: The purpose of this E-Letter is solely informational and educational. The information herein should not be considered to be a substitute for the direct medical advice of your doctor, nor is it meant to encourage the diagnosis or treatment of any illness, disease, or other medical problem by laypersons. If you are under a physician's care for any condition, he or she can advise you whether the information in this E-Letter is suitable for you. Readers should not make any changes in drugs, doses, or any other aspects of their medical treatment unless specifically directed to do so by their own doctors.

May 16, 2017 [The problem here is that the FDA are looking at things in isolation. There are unknown interactions in some people.]

Ongoing FDA Review of Fluoroquinolones Downplays Concerns Evidence Doesn't Support Link to Retinal Detachment, Aortic Problems

Bactericidal Antibiotics Induce Mitochondrial Dysfunction and Oxidative Damage in Mammalian Cells:

Abstract: Prolonged antibiotic treatment can lead to detrimental side effects in patients, including ototoxicity, nephrotoxicity, and tendinopathy, yet the mechanisms underlying the effects of antibiotics in mammalian systems remain unclear. It has been suggested that bactericidal antibiotics induce the formation of toxic reactive oxygen species (ROS) in bacteria. We show that clinically relevant doses of bactericidal antibiotics—quinolones, aminoglycosides, and β-lactams—cause mitochondrial dysfunction and ROS overproduction in mammalian cells. We demonstrate that these bactericidal antibiotic–induced effects lead to oxidative damage to DNA, proteins, and membrane lipids. Mice treated with bactericidal antibiotics exhibited elevated oxidative stress markers in the blood, oxidative tissue damage, and up-regulated expression of key genes involved in antioxidant defense mechanisms, which points to the potential physiological relevance of these antibiotic effects. The deleterious effects of bactericidal antibiotics were alleviated in cell culture and in mice by the administration of the antioxidant N-acetyl-L-cysteine or prevented by preferential use of bacteriostatic antibiotics. This work highlights the role of antibiotics in the production of oxidative tissue damage in mammalian cells and presents strategies to mitigate or prevent the resulting damage, with the goal of improving the safety of antibiotic treatment in people.

17β-Estradiol protects against apoptosis induced by levofloxacin in rat nucleus pulposus cells by upregulating integrin α2β1:

Abstract: Levofloxacin has been reported to have cytotoxicity to chondrocytes in vitro. And 17β-estradiol has been widely studied for its protective effects against cell apoptosis. Based on apoptotic cell model induced by levofloxacin, the purpose of this study was to explore the mechanism by which 17β-estradiol protects rat nucleus pulposus cells from apoptosis. Inverted phase-contrast microscopy, flow cytometry, and caspase-3 activity assay were used to find that levofloxacin induced marked apoptosis, which was abolished by 17β-estradiol. Interestingly, estrogen receptor antagonist, ICI182780, and functional blocking antibody to α2β1 integrin, both prohibited the effect of 17β-estradiol. Simultaneously, levofloxacin decreased cellular binding ability to type II collagen, which was also reversed by 17β-estradiol. Furthermore, western blot and real-time quantitative PCR were used to find that integrin α2β1 was responsible for estrogen-dependent anti-apoptosis, which was time–response and dose–response effect. 17β-estradiol was proved for the first time to protect rat nucleus pulposus cells against levofloxacin-induced apoptosis by upregulating integrin α2β1 signal pathway.

DNA-damage caused by antibiotic drugs - quinolones:

Abstract: In the present investigation, the genotoxic potencies (SOSIP) of 10 antibiotic quinolones (topoisomerase LT inhibitors) were tested in the sfiA::lacZ fusion containing strain Escherichia coli PQ37 using a modified procedure of the SOS chromotest. A number of quinolones exhibited extremely high DNA damaging effects in the absence of an exogenous metabolizing system. The highest SOS inducing potencies (SOSIP) exhibited sparfloxacin with 2,400 Delta IF/nmole, ciprofloxacin (SOSIP = 184 Delta IF/nmole) and norfloxacin (SOSIP=120 Delta IF/nmole), whereas pipemic acid (SOSIP=4.6 Delta IF/nmole), cinoxacin (SOSIP=0.5 Delta IF/nmole) and nalidixic acid (SOSIP=0.5 Delta IF/nmole) showed only weak genotoxicity. The possibility of mutagenic effects caused by quinolones in eukaryotic cells is discussed.

Cipro and Gulf War Syndrome:

… Cipro’s side effects turn out to be very similar to many of the illnesses that Gulf War veterans are experiencing due to Gulf War Syndrome. Cipro causes damage to liver enzymes which, when sufficiently depleted, destroy DNA. The death of DNA causes effects similar to autoimmune diseases in our bodies. The symptoms of the process of this cell death (oxidative stress and mitochondrial injury {OSMI}) are all found in the illnesses attributed to Gulf War Syndrome (Golomb, 2012). …

Ciprofloxacin-induced DNA damage in primary culture of rat astrocytes and protection by Vitamin E:

Abstract: Fluoroquinolones are generally well-tolerated antibiotics in patients. Gastrointestinal, central nervous system, and dermatological adverse events were the most frequent unwanted effects during therapy with these drugs. However, the mechanism of these adverse effects has not yet been elucidated. The aim of this study was to investigate the possible DNA damage-inducing effect of a fluoroquinolone (FQ) antibiotic, ciprofloxacin (CPFX) on primary culture of rat astrocytes. For this purpose, the cultured cells were incubated with various concentrations of CPFX, and DNA damage was monitored by comet assay. Our results showed a concentration-dependent induction of DNA damage by CPFX. Pretreatment of cells with Vitamin E for 4 h provided partial protection against this effect. The data obtained in this study suggest that CPFX-induced DNA damage might be related to oxidative stress and should be considered for further mechanistic studies of central nervous system toxicity of CPFX.

George Lesher discovered Quinolone's in 1962. – Laughing Gas, Viagra, and Lipitor: The Human Stories behind the Drugs We Use By Jie Jack Li.

Quinolones may cause severe blood sugar swings

Warning: Fluoroquinolone Antibiotics May Cause Permanent Nerve Damage

http://en.wikipedia.org/wiki/Adverse_effects_of_fluoroquinolones

These people should be in jail for the rest of their lives!:

“…Commonly used FQs include levofloxacin (Levaquin), ciprofloxacin (Cipro), moxifloxacin (Avelox), norfloxacin (Noroxin), ofloxacin (Floxin) and gemifloxacin (Factive).

…

For many people, the concept of being “floxed” is difficult to comprehend. “The reaction is mostly disbelief,” says Girard. “They have been taking antibiotics their whole life and never had anything like that. Or they have taken Cipro and they have arthritis or depression but don’t understand it could be from the Cipro. But the reaction is mostly disbelief - until it happens to you.”

Fluoroquinolones, or more accurately their precursors, were developed as a chemotherapy drug. When it was discovered that the compound could actually kill bacteria, they were reformulated and marketed as antibiotics. However, they are far from penicillin.

Simply put, fluoroquinolones work by entering the bacteria’s DNA and preventing it from reproducing. However, they do not work only on bacterial DNA. They actually penetrate all the cells in the body. It’s believed that the tendons rupture because the FQs confuse the DNA, and the cells literally get mixed up.

“Tendons will be rebuilding wrong, and rebuilding wrong until one day you overextend them and they snap. People get it in their shoulder, rotator cuff, hands, wrist,” adds Girard. … ” – Mark Girard; Fluoroquinolone Toxicity Group

Uncategorized:

FDA Allows Chemo Drugs To Be Prescribed As Antibiotics

“A few popular antibiotics affect DNA, similar to some chemotherapy agents. If you’re sensitive to them, you could pay a neurological price that causes sudden and serious neuropathy and degrees of brain damage. The Food and Drug Administration is concerned about drugs in the fluoroquinolone class, and these already have a black box warning for an increased risk of tendon ruptures. But I’m telling you that more reports have come in with accusations of neurological damage.” - Suzy Cohen, RPh

Certain Antibiotics Spur Widening Reports of Severe Side Effects

Next, a Health Unit report about a medical mystery, and the questions it’s raising about the drug-monitoring system. It involves a class of antibiotic drugs that some people say are making them very ill. – Judy Woodruff

Are Dangerous Antibiotics Causing Chronic Illness? by Lisa Bloomquist. Lisa hosts The Floxie Hope Podcast, also via iTunes, from Floxie Hope.

The Fluoroquinolone Toxicity Solution is Your Path to Recovery eBook updated for 2014.

The Harmful Effects of Antibiotics on the Human Microbiome.

Fluoroquinolone Wall of Pain.

Fluoroquinolone Mitochondrial Toxicity: FDA Petition. Write In, Be Heard! by My Quin Story.

Doctors with positive reviews for FQ Syndrome (informally called "FQ Toxicity") Please note that I do not maintain this list. Please read the list via the link and do not save, re-post or re-distribute, because the list can change frequently. For any questions regarding the list there is an email address listed on it. Courtesy of My Quin Story.

How Many Doctors Does it Take to Fix the Shower? A Tale of Fluoroquinolone Injury.

The Fluoroquinolone Antibiotic Side Effects Survey.

Tell Your Doctors: Stop Overprescribing Cipro, Levaquin & Other Fluoroquinolones.

Fluoroquinolone prescribing in the United States: 1995 to 2002.

Drug Watch has a page on Fluoroquinolones:

Drugwatch.com advocates for people to make a stand against prescription drugs that have harmful side effects and medical devices that cause damage. We do this by providing accurate, comprehensive information about these commonly used drugs and devices and by answering questions raised by people who contact us.

Our mission is to help our users evaluate if they have a legal case because of life-changing side effects or complications that were caused by a device maker or a drug company. We are sponsored by The Peterson Firm, a Washington, D.C., law firm, and we use the legal team at The Peterson Firm to educate these users about their circumstances. If a user expresses interest in speaking with a lawyer about bringing a legal case, a Patient Advocate may suggest a law firm to work with The Peterson Firm on the case. If a user retains the firms to handle his or her case, both firms will work on a contingency fee basis, with no costs or fees charged unless a recovery is made. Your Patient Advocate can explain the process in more detail.

Facebook Support Groups: [There are actually over 87 of them now, these are the main ones of the ones that I follow]

Fluoroquinolone Toxicity Group

Fluoroquinolone Toxicity 24/7 Live Chat Group

Floxed Lymies

Quinolone Vigilance Foundation is part of Safter Pills dot Org.

News Coverage [There have now been over 150 main stream media stories]:

Clark Howard Says near-fatal disease possibly caused by popular antibiotic [Cipro] – “I felt like death…Why would I want to be dead? … The generic Lipitor acted as a catalyst. That caused the supposed problems with Cipro to magnify and give me the rhabdomyolysis.”

Fluoroquinolone damage is even making the main stream press now it has gotten so bad!

Your antibiotic might blind you, cripple you for life, or even kill you.

Powerful Antibiotic Could Be A Prescription For Danger